Analysis of Dr. Teppema and Dr. Dahan's Work

Dr. Teppema and Dr. Dahan illustrated through their experiment that Hypoxia often caused the denervation of carotid bodies. This means that the affected carotid bodies did not harbor the ability to release neurotransmitters. The denervation would lead to the loss of the ability to detect an action potential by the carotid bodies. This action potential, or rather a difference in the voltage between the membrane of the intracellular and extracellular environment is what would originally cause the reaction by the chemoreceptive cells. If the action potential persists or grows without a restoration of the potential back to a restoration potential, that would lead to the loss of regulation to an extreme. This would directly affect the working state of the Hypoxic Drive by further disabling it and its consistency in performance. This would play a rather large role in the secondary development of the Hypoxic Drive under the carbon dioxide chemoreceptor driven respiratory system.  The scientists, Dr. Teppema and Dr. Dahan in fact displayed the concept that peripheral chemoreceptors in rats and other organisms were agents of decline in Hypoxic Ventilatory response. They charted the relative Hypoxic Ventilatory response over time in controlled experiments and marked the marginal decline of the response. This contends to the idea that the Hypoxic Drive has a tendency to lose its clutch on regulation. This is a grand reason for carbon dioxide regulation to peak and since it is a favorable trait to organisms, it would ideally grow as a genotype.

 

The image above is actually a representation of action potentials that are crucial to the body and the levels at which they operate.